Preliminary experiments show conolidine may inhibit particular ion channels, lowering neuronal excitability and limiting agony alerts. This mechanism is especially appropriate in neuropathic pain, where by abnormal signaling results in persistent discomfort. In addition, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to ache perception. Computational ... https://bobo590sww4.blogdiloz.com/profile